Skip to main content
Menu
Language
Appearance
ABOUT / OUR APPROACH

Evidence first, plain language always

How this resource sources, weighs, and translates hereditary-cancer testing science — from peer-reviewed registries to ACMG variant criteria — without a single product to sell.

Two laboratories can read the same variant and reach different conclusions. Our job is to show readers where the evidence is settled, where it is still moving, and how to tell the difference.
PROMPT Registry · research desk
~38.6%
of PROMPT-catalogued variants were of uncertain significance (VUS)
JCO / PROMPT
11%
of participants carried a variant with conflicting lab interpretations
JCO 2017, PMC5562435
5
ACMG/AMP classification tiers, from benign to pathogenic
ACMG/AMP 2015
~98%
genotyping success rate from a single saliva self-collection
DNA Genotek / PMC3422993

How a claim becomes a page

Every explainer on this site moves through the same editorial method — the same path a sample takes from swab to sequencer, applied to evidence instead of DNA.

  1. 01

    1 · Source from primary literature

    We start with peer-reviewed work and guideline bodies — NCI, ACMG/AMP, NCCN, NHGRI, and ASCO/JCO publications such as the PROMPT registry itself. Press releases and vendor copy are never a primary source.

  2. 02

    2 · Weigh the evidence level

    Risk figures are reported as ranges with their study context, not single hero numbers. We note sample size, population, and whether a finding is a consensus guideline or a single cohort.

  3. 03

    3 · Translate to plain language

    Technical tokens — gene names like PALB2, variant IDs like rs80357906, kit codes like OG-500 — are kept exact but explained in plain words on first use.

  4. 04

    4 · Separate education from decision

    We explain what tests measure and what terms mean. We never recommend a personal course of action — that belongs with a clinician or genetic counsellor who knows your history.

  5. 05

    5 · Cite, date, and revisit

    Each page carries numbered references with links. Variant science changes; pages note when a figure reflects a point-in-time classification rather than a fixed fact.

What we report, and how we qualify it

A worked example of the editorial standard: the same hereditary-breast-cancer genes, with the qualifiers we attach so a range is never mistaken for a verdict.

GeneRisk tierReported lifetime rangeHow we qualify it
BRCA1 / BRCA2High penetrance~60–80%Cite as a range; flag dependence on family history and ascertainment
PALB2High penetranceSimilar magnitude to BRCA2State the comparison, not a fixed percent, where sources do
CHEK2Moderate penetrance~2–3× baselineExpress as relative risk; avoid implying a single absolute number
ATMModerate penetranceElevated; estimates evolvingMark as an area of active research; date the estimate

Table 1. Illustrative lifetime breast-cancer risk by gene, with editorial qualifiers. Figures are population estimates that vary by study, family history, and ancestry — not individual predictions.

Why variant interpretation needs careful sourcing

From the PROMPT registry's own catalogue — the spread of classifications shows why we report uncertainty honestly rather than rounding it away. A large share of variants are simply not yet resolved.

Pathogenic36.2 %

Clinically actionable

VUS (uncertain)38.6 %

Not yet resolved — the largest group

Conflicting interpretation14.4 %

Labs disagree

Benign / likely benign3.5 %

Reassuring

Key terms, defined the way we use them

A few load-bearing terms. Full definitions live in the glossary; these are the ones the method depends on.

VUS
Variant of uncertain significance — a change in a gene such as CHEK2 whose effect on cancer risk is not yet established. We treat a VUS as a question, never as a diagnosis.
Penetrance
The proportion of people carrying a variant who actually develop the associated condition. High-penetrance genes like BRCA1 differ sharply from moderate-penetrance genes like ATM.
ACMG/AMP tiers
The five-tier framework (pathogenic, likely pathogenic, uncertain, likely benign, benign) published by the American College of Medical Genetics and the Association for Molecular Pathology in 2015, built from 28 weighted evidence criteria.
Multiplex / panel testing
Sequencing several genes at once — e.g. ATM, CHEK2, PALB2, BRIP1 — rather than one gene in isolation. The basis of the PROMPT registry's data.
Analytic vs. clinical validity
Whether a test reliably detects what it claims (analytic) versus whether the result meaningfully predicts an outcome (clinical). We keep the two distinct.

Our stance, in plain terms

Do you sell tests, kits, or sequencing services?

No. This is an editorial resource in its authority phase — there are no product links, affiliate codes, or shop pages anywhere on the site. Kit codes such as OG-500 appear only as technical references, not recommendations to buy.

Is the original PROMPT study still enrolling?

We make no such claim. PROMPT (Prospective Registry of Multiplex Testing) is a real multi-institution hereditary-cancer registry whose findings were published through ASCO/JCO. We honour that heritage and cite its data; we do not represent it as currently recruiting.

How do you handle conflicts of interest?

Our sourcing prioritises non-commercial guideline bodies (NCI, ACMG, NCCN, NHGRI). Where we cite a manufacturer's technical specification — for instance saliva DNA yield from a self-collection kit — we label it as a vendor specification, not independent evidence.

Is anything here medical advice?

No. These are educational explainers. For any personal decision about testing, results, or risk, speak with a clinician or a certified genetic counsellor who can review your individual and family history.

References
  1. [1]JCO / PROMPT 2017. Balmaña J, Digiovanni L, Gaddam P, et al. Conflicting Interpretation of Genetic Variants and Cancer Risk by Commercial Laboratories as Assessed by the Prospective Registry of Multiplex Testing. J Clin Oncol.
  2. [2]ASCO 2016. Walsh MF, et al. Prospective Registry of Multiplex Testing (PROMPT): a web-based platform to assess cancer risk of genetic variants. ASCO Annual Meeting abstract 1518.
  3. [3]ACMG/AMP 2015. Richards S, Aziz N, Bale S, et al. Standards and guidelines for the interpretation of sequence variants: ACMG and AMP joint consensus recommendation. Genet Med.
  4. [4]MSK PROMPT. Memorial Sloan Kettering Cancer Center. Genetic Counseling and Genetic Testing for Hereditary Cancer: The PROMPT Study.
  5. [5]DNA Genotek / PMC3422993. Quality of DNA extracted from saliva samples collected with the Oragene self-collection kit; DNA Genotek OG-500 technical specification.

Start with the evidence, not the marketing

Every guide on this site follows the method above — sourced, qualified, and free of anything for sale. Explore the explainers, or read how variant classification actually works.